Dylan Gee
· Assistant ProfessorVerifiedYale University · Department of Molecular, Cellular, and Developmental Biology
Active 2008–2026
About
Dr. Dylan Gee is a Professor in the Department of Psychology at Yale University. She earned her B.A. in Psychological and Brain Studies from Dartmouth College and her Ph.D. in Clinical Psychology from UCLA. Before joining Yale's faculty, Dr. Gee completed a clinical internship at Weill Cornell Medical College and a research fellowship at the Sackler Institute for Developmental Psychobiology. Her research centers on developmental psychopathology, aiming to delineate typical and atypical brain development. She investigates how early environments, including stress, trauma, and parenting, influence sensitive periods in neurodevelopment and contribute to risk for anxiety and stress-related disorders. Dr. Gee's work also focuses on translating knowledge of brain development to inform clinical interventions and policy related to youth mental health.
Research topics
- Psychology
- Medicine
- Political Science
- Psychiatry
- Developmental psychology
- Public relations
- Business
- Pathology
- Neuroscience
- Clinical psychology
- Internal medicine
- Nursing
- Social psychology
- Audiology
- Engineering
- Family medicine
- Psychoanalysis
- Cognitive psychology
- Engineering ethics
- Pediatrics
- Physiology
- Psychotherapist
- Applied psychology
- Virology
Selected publications
Parent-child neural similarity during socioemotional processing relates to internalizing symptoms
2026-04-19
articleOpen accessIntergenerational transmission of mental well-being and socioemotional functioning is widely documented at the behavioral level, yet how such patterns are instantiated in the brain remains poorly understood. Using a movie-watching fMRI paradigm, we investigated parent-child similarity in socioemotional processing and its associations with family environment, parental emotion regulation, and similarity in internalizing symptoms. Across 258 family members (120 children, 138 parents), parent–child dyads showed greater neural similarity in socioemotional processing than unrelated adult–child pairs. Effects were concentrated in prefrontal and temporal regions, particularly the right lateral prefrontal cortex (PFC). Neural similarity was stronger in mother–daughter and father–son dyads. Poorer family functioning was associated with reduced whole-brain similarity. Critically, parent-child right lateral PFC similarity was associated with similarity in internalizing symptoms depending on parental emotion regulation. These findings establish parent–child socioemotional neural similarity as a context-dependent pathway through which intergenerational patterns of mental well-being may emerge.
Developmental Cognitive Neuroscience · 2026-01-28
articleOpen accessThis review synthesizes ten years of research utilizing data from the Adolescent Brain Cognitive Development (ABCD) Study, emphasizing how the study's comprehensive, longitudinal design supports a multivariate understanding of adolescent mental health. We focus on studies that have examined the collective or interacting relations of multiple factors to mental health in adolescents, as this unique dataset allows for examining more complex configurations of risk factors. We highlight key findings from ABCD data that have deepened our understanding of the risk factors shaping mental health outcomes in adolescence. Findings underscore the complex interplay of biological, psychological, and/or contextual factors on adolescent mental health. We conclude with a forward-looking discussion of emerging research priorities and opportunities to further leverage the ABCD dataset to inform developmental theory, prevention, and intervention efforts.
Social Threat and Childhood Security
OSF Preprints (OSF Preprints) · 2026-02-27
otherChild Psychiatry & Human Development · 2025-01-09 · 9 citations
articleSenior authorBiological Psychiatry Global Open Science · 2025-04-17 · 4 citations
articleOpen accessBackground: It is unclear how transdiagnostic symptoms including attention, disruptive behavior, and internalizing problems are linked to in-scanner motion in children across structural and functional magnetic resonance imaging (fMRI). In the current study, we examined whether transdiagnostic symptoms of attention, disruptive behavior, and internalizing problems were associated with scanner motion in children during multimodal imaging. Methods: In 9045 children ages 9 to 10 years in the ABCD (Adolescent Brain Cognitive Development) Study, logistic regression and linear mixed-effects models were used to examine associations between motion and behavior. Motion was indexed using ABCD Study quality control (QC) metrics and mean framewise displacement for T1- and T2-weighted structural, resting-state, and diffusion MRI; stop-signal task; monetary incentive delay task; and emotional n-back task. The Child Behavior Checklist was used as a continuous measure of symptom severity. Results: Greater severity of attention and disruptive behavior problems was associated with a lower likelihood of passing motion QC across imaging modalities, while increased internalizing severity was associated with a higher likelihood of passing. There was also an interaction between sex and attention-related problems in passing QC for T2-weighted and diffusion MRI scans. Increased attention and disruptive behavior problems were associated with increased mean motion, whereas increased internalizing problems were associated with decreased mean motion. Greater severity of attention problems was associated with worse performance across the fMRI tasks. Conclusions: These findings have implications for advancing the development of computational and behavioral approaches for mitigating motion effects in youths, enhancing accessibility of imaging protocols and representativeness influences across child psychiatric disorders, and identifying brain-based biomarkers.
Dynamic Resting-State Network Markers of Disruptive Behavior Problems in Youth
bioRxiv (Cold Spring Harbor Laboratory) · 2025-05-20
preprintOpen accessBackground: Childhood disruptive behavior problems are linked to aberrant integrity within large-scale cognitive control networks. However, it is unclear if transitory or dynamic variation in the functional brain architecture is a marker of disruptive behavior problems. The current study tested whether functional connectivity across dynamic networks is distinctly associated with the transdiagnostic symptom domain of disruptive behavior problems in children. Methods: Participants were aged 9-10 years from the Adolescent Brain Cognitive Development (ABCD) Study, who completed resting-state fMRI (N=877). We employed a dynamic connectivity approach leveraging a hidden semi-Markov model (HSMM) to identify transient properties of brain networks and states. Models estimated the time spent in each state (occupancy time) and the number of consecutive timepoints in a state (dwell time) for each participant. Linear regression models were utilized to identify distinct associations between dynamic properties (occupancy and sojourn times) and severity of disruptive behavior problems, accounting for other commonly co-occurring symptoms. Results: Dynamic network markers of disruptive behavior problems included increased time in network states characterized by globally aberrant connectivity patterns in circuitry involved in cognitive control including frontoparietal and dorsal attention networks. Replication of findings was found in a held-out sample of resting-state fMRI runs in which greater severity of disruptive behavior problems was uniquely linked to greater occupancy time in similarly characterized brain states. Conclusion: Transdiagnostic, dynamic resting-state markers of disruptive behavior problems in youth may assist in the development of brain-based biomarkers for monitoring treatment outcomes, assessing circuit target engagement and informing clinical decisions.
UNC Libraries · 2025-05-13
articleOpen accessCOVID-19 presents significant social, economic, and medical challenges. Because COVID-19 has already begun to precipitate huge increases in mental health problems, clinical psychological science must assert a leadership role in guiding a national response to this secondary crisis. In this article, COVID-19 is conceptualized as a unique, compounding, multidimensional stressor that will create a vast need for intervention and necessitate new paradigms for mental health service delivery and training. Urgent challenge areas across developmental periods are discussed, followed by a review of psychological symptoms that likely will increase in prevalence and require innovative solutions in both science and practice. Implications for new research directions, clinical approaches, and policy issues are discussed to highlight the opportunities for clinical psychological science to emerge as an updated, contemporary field capable of addressing the burden of mental illness and distress in the wake of COVID-19 and beyond.
Communications Psychology · 2025-03-05 · 3 citations
articleOpen accessSenior authorParsing heterogeneity in the nature of adversity exposure and neurobiological functioning may facilitate better understanding of how adversity shapes individual variation in risk for and resilience against anxiety. One putative mechanism linking adversity exposure with anxiety is disrupted threat and safety learning. Here, we applied a person-centered approach (latent profile analysis) to characterize patterns of adversity exposure at specific developmental stages and threat/safety discrimination in corticolimbic circuitry in 120 young adults. We then compared how the resultant profiles differed in anxiety symptoms. Three latent profiles emerged: (1) a group with lower lifetime adversity, higher neural activation to threat, and lower neural activation to safety; (2) a group with moderate adversity during middle childhood and adolescence, lower neural activation to threat, and higher neural activation to safety; and (3) a group with higher lifetime adversity exposure and minimal neural activation to both threat and safety. Individuals in the second profile had lower anxiety than the other profiles. These findings demonstrate how variability in within-person combinations of adversity exposure and neural threat/safety discrimination can differentially relate to anxiety, and suggest that for some individuals, moderate adversity exposure during middle childhood and adolescence could be associated with processes that foster resilience to future anxiety.
Psychological Trauma Theory Research Practice and Policy · 2025-08-14 · 1 citations
articleOpen accessSenior authorOBJECTIVE: Early-life stress (ELS) has been linked to the development of psychopathology-related outcomes, including posttraumatic stress symptoms (PTSS). Locus of control (LOC) is one factor thought to be critical to the development of PTSS. METHOD: = 665) to identify subgroups based on LOC orientation and PTSS severity and to explore early environmental correlates of individuals' membership in distinct subgroups. RESULTS: Across both samples, results of the latent profile analyses indicated a best fitting five-class solution. Cumulative ELS and perceived uncontrollability of ELS exposure emerged as significant predictors of subgroup membership in the Mechanical Turk sample. CONCLUSIONS: These findings suggest that associations between perceptions of control at the event (i.e., controllability during a stressful event) and global (i.e., LOC orientation) levels may play a role in the development of stress-related symptoms following ELS. (PsycInfo Database Record (c) 2025 APA, all rights reserved).
Biological Psychiatry Cognitive Neuroscience and Neuroimaging · 2025-02-18 · 4 citations
articleOpen accessSenior authorCorresponding
Recent grants
7/21 ABCD-USA CONSORTIUM: RESEARCH PROJECT SITE AT YALE
NIH · $19.7M · 2015–2027
CAREER: Neurodevelopmental Mechanisms of Stressor Controllability and Resilience
NSF · $706k · 2022–2027
Novel Mechanisms of Fear Reduction Targeting the Biological State of the Developing Brain
NIH · $1.3M · 2015–2020
Frequent coauthors
- 77 shared
Nim Tottenham
Columbia University
- 57 shared
Alexis Brieant
University of Vermont
- 51 shared
Hajer Nakua
Centre for Addiction and Mental Health
- 50 shared
Anna Vannucci
Columbia University
- 50 shared
divya brundavanam
Karolinska Institutet
- 50 shared
Jenny Harris
- 50 shared
Jack R Lovell
University of Colorado Boulder
- 40 shared
Emily M. Cohodes
Yale University
Labs
CANDLabPI
Focuses on developmental psychopathology, delineating typical and atypical brain development, and translating knowledge of brain development to inform clinical interventions and policy related to youth mental health.
Education
B.A., Psychological & Brain Sciences
Dartmouth College
Ph.D., Psychology
University of California Los Angeles
Awards & honors
- NIH Director’s Early Independence Award
- NARSAD Young Investigator Award
- Jacobs Foundation Early Career Award
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